c-Myc transactivation domain-associated kinases: questionable role for map kinases in c-Myc phosphorylation

J Cell Biochem. 1999 Mar 15;72(4):483-91. doi: 10.1002/(sici)1097-4644(19990315)72:4<483::aid-jcb4>3.0.co;2-i.

Abstract

We have isolated and characterized cellular kinases which associate with the transactivation domain of c-Myc and phosphorylate Ser-62. We demonstrate that cellular Map kinases associate with c-Myc under stringent conditions and phosphorylate Ser-62. We also find that TPA stimulates the activity of the Myc-associated Map kinase to phosphorylate Ser-62. However, we do not observe an increase in Ser-62 phosphorylation in endogenous c-Myc after TPA treatment of cells. Since the regulation of the c-Myc-associated Map kinases does not correlate with the in vivo regulation of Ser-62 phosphorylation in c-Myc, we conclude that Map kinases are not the in vivo kinases for Ser-62. Although Ser-62 phosphorylation was not affected by TPA, phosphorylation at a different serine residue was significantly upregulated by TPA.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 3T3 Cells
  • Animals
  • COS Cells
  • Calcium-Calmodulin-Dependent Protein Kinases / metabolism*
  • Mice
  • Phosphopeptides / analysis
  • Phosphorylation
  • Phosphoserine / analysis
  • Protein Kinases / metabolism*
  • Proto-Oncogene Proteins c-myc / metabolism*
  • Tetradecanoylphorbol Acetate / pharmacology

Substances

  • Phosphopeptides
  • Proto-Oncogene Proteins c-myc
  • Phosphoserine
  • Protein Kinases
  • Calcium-Calmodulin-Dependent Protein Kinases
  • Tetradecanoylphorbol Acetate