IL-10 induces a Th2 cell tolerance in allergic asthma

Int Arch Allergy Immunol. 1999 Feb-Apr;118(2-4):391-4. doi: 10.1159/000024145.

Abstract

Background: Interleukin (IL)-10 induces a long-term antigen-specific anergy in human CD4+ T cells.

Methods: Peripheral blood mononuclear cells (PBMCs) were isolated from house dust mite (Dermatophagoides farinae, Der f)-sensitized asthmatic patients. PBMCs were stimulated with Der f antigen for 72 h immediately after purification or after 48 h of resting culture with medium, and IL-10 and IL-5 in the culture supernatant were measured. PBMCs were also stimulated with Der f antigen for 7 days either immediately after purification or after 48 h of resting culture, after which cells were collected. Secondary proliferative responses of these cells to stimulation for 3 days with Der f antigen and mitomycin C-treated PBMCs as antigen-presenting cells or with phorbol myristate acetate (PMA) plus calcium ionophore were investigated.

Results: Stimulation of PBMCs with Der f antigen immediately after purification significantly increased the proliferative response and IL-5 production. Stimulation of PBMCs with Der f antigen after resting culture with medium alone for 48 h significantly decreased IL-5 production and markedly increased IL-10 production. Although activation of cells with Der f antigen immediately after purification significantly increased secondary proliferative responses, stimulation after 48 h of resting culture failed to increase secondary proliferative responses. However, proliferation recovered when cells were activated with PMA plus calcium ionophore.

Conclusion: These results suggest that antigen-specific Th2 cells are anergized by IL-10 and that Th2 cell tolerance may suppress eosinophilic inflammation in allergic asthma.

MeSH terms

  • Allergens / immunology*
  • Animals
  • Cells, Cultured
  • Dust
  • Humans
  • Immune Tolerance / drug effects
  • Interleukin-10 / immunology*
  • Interleukin-10 / pharmacology
  • Lymphocyte Activation / drug effects
  • Mites
  • Th2 Cells / immunology*

Substances

  • Allergens
  • Dust
  • Interleukin-10