Analysis of Epstein-Barr virus (EBV) receptor CD21 on peripheral B lymphocytes of long-term EBV- adults

Clin Exp Immunol. 1999 Jun;116(3):468-73. doi: 10.1046/j.1365-2249.1999.00912.x.

Abstract

Primary infections with EBV are rarely observed after the age of 20. Some individuals even remain seronegative all their lives. Previously, a lack of EBV receptors on B cells of persistently EBV- adults was described as a reason for long-term EBV-seronegativity. The present study examined the CD21 receptor status of 20 repeatedly EBV- healthy adults and 32 EBV+ volunteers by means of flow cytometry. CD21 molecules on the surface of CD19+ B cells were quantified using anti-IgG-coated microbeads. The percentage of CD19+/CD21+ B lymphocytes was slightly lower in the peripheral blood of EBV- donors, but the CD21 antibody binding capacity on CD19+ B cells showed no significant differences between EBV- and EBV+ adults. In vitro studies showed an equally good EBV transformability of peripheral B lymphocytes of EBV- and EBV+ donors. Since HLA-DR was recently described as a co-receptor for EBV infection of B cells, we also determined HLA-DRB1 alleles in the EBV- group. We found a significant negative association of EBV-seronegativity with HLA-DR13 in comparison with 111 healthy blood donors. In summary, a biologically significant lack of the EBV receptor CD21 on peripheral B lymphocytes of persistently EBV- adults was excluded as a reason for long-term EBV-seronegativity.

MeSH terms

  • Adult
  • Antibodies, Viral / blood
  • B-Lymphocytes / immunology*
  • Cell Transformation, Viral
  • Epstein-Barr Virus Infections / blood
  • Epstein-Barr Virus Infections / genetics
  • Epstein-Barr Virus Infections / immunology*
  • Female
  • Gene Frequency
  • HLA-DR1 Antigen / genetics
  • Herpesvirus 4, Human / immunology
  • Humans
  • In Vitro Techniques
  • Leukocyte Count
  • Male
  • Middle Aged
  • Monocytes / immunology
  • Receptors, Complement 3d / metabolism*

Substances

  • Antibodies, Viral
  • HLA-DR1 Antigen
  • Receptors, Complement 3d