Berberine-induced apoptosis of human leukemia HL-60 cells is associated with down-regulation of nucleophosmin/B23 and telomerase activity

Int J Cancer. 1999 Jun 11;81(6):923-9. doi: 10.1002/(sici)1097-0215(19990611)81:6<923::aid-ijc14>3.0.co;2-d.

Abstract

The steady-state level of nucleophosmin/B23 mRNA decreased during berberine-induced (25 microg/ml, 24 to 96 hr) apoptosis of human leukemia HL-60 cells. A decline in telomerase activity was also observed in HL-60 cells treated with berberine. A stable clone of nucleophosmin/B23 overexpressed in HL-60 cells was selected and found to be less responsive to berberine-induced apoptosis. About 35% to 63% of control vector-transfected cells (pCR3) exhibited morphological characteristics of apoptosis, while about 8% to 45% of nucleophosmin/B23-over-expressed cells (pCR3-B23) became apoptotic after incubation with 15 microg/ml berberine for 48 to 96 hr. DNA extracted from pCR3 cells contained more fragmented DNA than pCR3-B23 cells during treatment with 15 microg/ml berberine for 24 to 48 hr. Our results indicate that berberine-induced apoptosis is associated with down-regulation of nucleophosmin/B23 and telomerase activity. We also suggest that nucleophosmin/B23 may play an important role in the control of the cellular response to apoptosis induction.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects
  • Apoptosis / physiology*
  • Berberine / pharmacology*
  • Cell Division / drug effects
  • Cell Survival / drug effects
  • DNA Fragmentation
  • Dose-Response Relationship, Drug
  • Gene Expression Regulation, Neoplastic / drug effects*
  • HL-60 Cells / cytology
  • HL-60 Cells / drug effects*
  • HL-60 Cells / physiology
  • Humans
  • Kinetics
  • Nuclear Proteins / genetics*
  • Nucleophosmin
  • Phosphoproteins / genetics
  • Telomerase / genetics*
  • Time Factors

Substances

  • NPM1 protein, human
  • Nuclear Proteins
  • Phosphoproteins
  • Berberine
  • Nucleophosmin
  • Telomerase