Differential activity of the -2.7 kb chicken lysozyme enhancer in macrophages of different ontogenic origins is regulated by C/EBP and PU.1 transcription factors

DNA Cell Biol. 1999 Aug;18(8):631-42. doi: 10.1089/104454999315042.

Abstract

Expression of the chicken lysozyme gene is upregulated during macrophage maturation. Recently, an additional regulatory feature was discovered: the gene is differentially expressed in macrophages of embryonic/fetal and adult origin. The lysozyme gene is only weakly expressed in mature embryo-derived macrophages, whereas there is a high level of expression in macrophages derived from adult animals. This finding provided a molecular tool to investigate the heretofore ill-defined differences between embryonic/fetal- and adult-type macrophages. We showed that the low expression in the embryo is associated with reduced activity of the myeloid-specific -2.7 kb lysozyme enhancer. Our protein-binding analyses and transfection studies demonstrated that this enhancer, in order to be fully active in activated macrophages, requires the combined action of C/EBPs, PU.1, and a third, as yet unidentified, protein binding to an AP-1-like site. Of these three, PU.1 and C/EBPs display significantly reduced nuclear DNA-binding activities in embryo-derived macrophages compared with adult-type cells. These results point to different roles of C/EBPs and PU.1 in embryonic/fetal and adult myelopoiesis.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging
  • Animals
  • Bone Marrow Cells / drug effects
  • Bone Marrow Cells / enzymology
  • Bone Marrow Cells / metabolism
  • CCAAT-Enhancer-Binding Proteins
  • Cell Line
  • Chick Embryo
  • Chickens
  • DNA / genetics
  • DNA / metabolism
  • DNA-Binding Proteins / metabolism*
  • Enhancer Elements, Genetic / genetics*
  • Gene Expression Regulation, Developmental*
  • Lipopolysaccharides / pharmacology
  • Macrophage Activation
  • Macrophages / cytology
  • Macrophages / enzymology
  • Macrophages / metabolism*
  • Muramidase / genetics*
  • Mutation
  • Nuclear Proteins / metabolism*
  • Protein Isoforms / metabolism
  • Proto-Oncogene Proteins / metabolism*
  • Response Elements / genetics
  • Trans-Activators / metabolism*
  • Transfection

Substances

  • CCAAT-Enhancer-Binding Proteins
  • DNA-Binding Proteins
  • Lipopolysaccharides
  • Nuclear Proteins
  • Protein Isoforms
  • Proto-Oncogene Proteins
  • Trans-Activators
  • proto-oncogene protein Spi-1
  • DNA
  • Muramidase