[Activated protein C resistance in patients with central retinal vein occlusion in comparison to patients with a history of deep-vein thrombosis and a healthy control group]

Ophthalmologe. 1999 Sep;96(9):594-9. doi: 10.1007/s003470050458.
[Article in German]

Abstract

Background: The recently described "APC resistance" caused by a mutant form of factor V (factor V Leiden) is the most frequent cause of hereditary thrombosis. This study was carried out to investigate the association between activated protein C resistance and central retinal vein occlusion (CRVO). We evaluated the prevalence of APC resistance in patients with CRVO, patients with a history of deep-vein thrombosis, and a healthy control group.

Patients and methods: We examined 107 patients with CRVO, 112 patients with deep-vein thrombosis and 70 healthy individuals. The test performed was a modified APC-resistance assay using factor V-deficient plasma.

Results: We identified APC resistance in 5.6 % of patients with CRVO and in 5.7 % of the control group. All carriers were heterozygous. In the deep-vein thrombosis group 23.2 % tested positive for APC resistance. Four patients were homozygous and 22 were heterozygous carriers.

Conclusion: These results indicate that APC resistance has no major role in the pathogenesis of CRVO. Routine testing for the presence of factor V Leiden mutant in CRVO is not necessary.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Factor V / genetics
  • Female
  • Genetic Carrier Screening
  • Humans
  • Male
  • Middle Aged
  • Partial Thromboplastin Time
  • Retinal Vein Occlusion / blood
  • Retinal Vein Occlusion / diagnosis
  • Retinal Vein Occlusion / genetics*
  • Thrombophlebitis / blood
  • Thrombophlebitis / diagnosis
  • Thrombophlebitis / genetics*

Substances

  • factor V Leiden
  • Factor V