Differential effect of estradiol on antibody secretion of murine hybridomas

Hybridoma. 1999 Aug;18(4):377-83. doi: 10.1089/hyb.1999.18.377.

Abstract

The need for increased antibody production by hybridomas has been approached by the addition to cell cultures of different growth factors; in vitro addition of estradiol-17beta (E2) to human blood lymphocytes increases the accumulation of plasma-blasts and Ig-secreting cells. Four different murine-murine hybridomas secreting different monoclonal antibodies (MAbs) were treated with E2. Specific antibody concentration was measured by enzyme-linked immunoadsorbent assay (ELISA) in culture supernatants whereas expression of E2-receptor in the hybridoma cells was determined by polymerase chain reaction (PCR). When E2 was added as a growth supplement to alpha-estrogen receptor positive murine-murine hybridomas it enhanced MAb secretion by as much as 255%, in a dose-dependant manner. This effect lasted for as long as the alpha-estrogen receptor was detected in the hybridoma cells, was inhibited by tamoxifen and was not observed in alpha-estrogen receptor negative hybridomas. The synthetic estrogen analogue diethylstilbestrol had no effect. Estradiol-17beta should be added to the list of hybridoma-inducing growth factors.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibody Formation / drug effects*
  • Antibody-Producing Cells / chemistry
  • B-Lymphocytes / chemistry
  • B-Lymphocytes / drug effects
  • B-Lymphocytes / immunology
  • Diethylstilbestrol / immunology
  • Diethylstilbestrol / pharmacology
  • Estradiol / immunology*
  • Estradiol / pharmacology*
  • Female
  • Hybridomas / chemistry
  • Hybridomas / drug effects*
  • Hybridomas / immunology*
  • Immunoglobulin G / drug effects
  • Male
  • Mice
  • Receptors, Estrogen / analysis
  • Receptors, Estrogen / immunology

Substances

  • Immunoglobulin G
  • Receptors, Estrogen
  • Estradiol
  • Diethylstilbestrol