Clinical, pharmacokinetic, and pharmacodynamic effects of tolcapone withdrawal in levodopa-treated patients with parkinsonism

Clin Neuropharmacol. 2000 Mar-Apr;23(2):98-105. doi: 10.1097/00002826-200003000-00007.

Abstract

The effect and clinical significance of tolcapone withdrawal on erythrocyte catechol-O-methyltransferase (COMT) activity, levodopa pharmacokinetics, and levodopa requirements were investigated in 59 patients with fluctuating parkinsonism who were randomized to receive placebo or tolcapone 100 or 200 mg three times daily for 6 weeks. Tolcapone withdrawal caused a transient elevation in COMT activity by 64% in patients receiving 100 mg three times daily and by 128% in those receiving 200 mg three times daily at approximately 1-2 weeks after discontinuation of drug. Thereafter, COMT activity was declining but did not reach baseline values by the 12-week study endpoint. However, this had no effect on plasma levodopa and 3-O-methyldopa (3-OMD) concentrations or on levodopa requirements. During treatment, tolcapone increased "on" time and decreased "off" time; after discontinuation of study medication and levodopa dose adjustment, on and off times were similar to baseline. Withdrawal was generally well tolerated; no patients withdrew from the trial during the posttreatment period, and no serious adverse events were observed. In conclusion, the transient increase in erythrocyte COMT activity observed after discontinuation oftolcapone is not associated with changes in peripheral levodopa metabolism and therefore has no significant clinical consequences in terms of levodopa requirements, clinical symptoms, or adverse events.

Publication types

  • Clinical Trial
  • Multicenter Study
  • Randomized Controlled Trial

MeSH terms

  • Aged
  • Antiparkinson Agents / adverse effects*
  • Antiparkinson Agents / blood
  • Antiparkinson Agents / therapeutic use*
  • Benzophenones / adverse effects*
  • Benzophenones / pharmacokinetics
  • Catechol O-Methyltransferase / blood
  • Double-Blind Method
  • Erythrocytes / drug effects
  • Erythrocytes / enzymology
  • Female
  • Humans
  • Levodopa / blood
  • Levodopa / pharmacokinetics
  • Levodopa / therapeutic use*
  • Male
  • Nitrophenols
  • Parkinson Disease / drug therapy
  • Parkinson Disease / enzymology
  • Parkinson Disease / physiopathology*
  • Substance Withdrawal Syndrome / enzymology
  • Substance Withdrawal Syndrome / physiopathology*
  • Tolcapone
  • Tyrosine / analogs & derivatives
  • Tyrosine / blood
  • Up-Regulation / drug effects

Substances

  • Antiparkinson Agents
  • Benzophenones
  • Nitrophenols
  • Tyrosine
  • Levodopa
  • Tolcapone
  • Catechol O-Methyltransferase
  • 3-methoxytyrosine