Details of hyperplastic polyps of the stomach shrinking after anti-Helicobacter pylori therapy

J Gastroenterol. 2000;35(5):372-5. doi: 10.1007/s005350050363.

Abstract

The precise etiology of hyperplastic polyps of the stomach is unknown, but recent studies suggest that they arise as a consequence of inflammation occurring in intimate association with Helicobacter pylori infection. The process of polyp regression after anti-H. pylori therapy, however, is unclear. Here we report a patient with large hyperplastic polyps of the stomach that regressed markedly after anti-H. pylori therapy. Histological examination of the regressed polyps revealed a decrease in the height of the hyperplastic foveolar epithelium and a decrease in the amount of inflammatory cell infiltration in the stroma. In addition, the percentage of Ki-67-positive hyperplastic epithelial cells markedly decreased after anti-H. pylori therapy, indicating that the epithelial cell proliferation rate had markedly decreased after treatment. At the same time, the degree of cyclooxygenase-2 expression in epithelial cells in the polyps decreased after treatment. Because cyclooxygenase-2 is expressed at sites of inflammation or neoplasm, these findings are consistent with a decrease in inflammatory cell infiltration, and represent resolving inflammation.

Publication types

  • Case Reports

MeSH terms

  • Anti-Bacterial Agents*
  • Anti-Ulcer Agents / therapeutic use*
  • Biomarkers, Tumor
  • Cell Division
  • Cyclooxygenase 2
  • Drug Therapy, Combination / therapeutic use*
  • Gastroscopy
  • Helicobacter Infections / complications
  • Helicobacter Infections / drug therapy*
  • Helicobacter Infections / pathology
  • Humans
  • Hyperplasia
  • Isoenzymes / metabolism
  • Ki-67 Antigen / metabolism
  • Male
  • Membrane Proteins
  • Middle Aged
  • Omeprazole / therapeutic use*
  • Polyps / complications
  • Polyps / drug therapy
  • Polyps / metabolism
  • Polyps / pathology*
  • Prostaglandin-Endoperoxide Synthases / metabolism
  • Stomach Neoplasms / complications
  • Stomach Neoplasms / drug therapy
  • Stomach Neoplasms / metabolism
  • Stomach Neoplasms / pathology*

Substances

  • Anti-Bacterial Agents
  • Anti-Ulcer Agents
  • Biomarkers, Tumor
  • Isoenzymes
  • Ki-67 Antigen
  • Membrane Proteins
  • Cyclooxygenase 2
  • PTGS2 protein, human
  • Prostaglandin-Endoperoxide Synthases
  • Omeprazole