Oxidized-LDL and atherosclerosis. Role of LOX-1

Ann N Y Acad Sci. 2000 May:902:95-100; discussion 100-2. doi: 10.1111/j.1749-6632.2000.tb06304.x.

Abstract

The accumulation of substantial numbers of monocyte/macrophages and activated T lymphocytes in focal areas of the arterial intima appears to be a hallmark of atherosclerosis. Our report demonstrated that lysophosphatidylcholine (lyso-PC), a polar phospholipid component that is increased in atherosclerotic lipoproteins, such as oxidized LDL and remnant lipoproteins in diabetic and Type 3 hyperlipidemia, can upregulate adhesion molecules for monocytes and T lymphocytes, and growth factors, such as heparin-binding epidermal growth factor-like growth factor and PDGF A and B chains. Recently, we identified the novel receptor for oxidized LDL, named LOX-1. We summarize the importance of the interaction between oxidized LDL and its receptor, LOX-1, in terms of early stage atherogenesis.

Publication types

  • Review

MeSH terms

  • Animals
  • Arteriosclerosis / pathology
  • Arteriosclerosis / physiopathology*
  • Humans
  • Lipoproteins, LDL / blood*
  • Macrophages / physiology
  • Receptors, LDL / physiology*
  • Receptors, Oxidized LDL
  • Scavenger Receptors, Class E
  • T-Lymphocytes / physiology

Substances

  • Lipoproteins, LDL
  • OLR1 protein, human
  • Receptors, LDL
  • Receptors, Oxidized LDL
  • Scavenger Receptors, Class E
  • oxidized low density lipoprotein