Reovirus-induced sigma1s-dependent G(2)/M phase cell cycle arrest is associated with inhibition of p34(cdc2)

J Virol. 2001 Aug;75(16):7429-34. doi: 10.1128/JVI.75.16.7429-7434.2001.

Abstract

Serotype 3 reoviruses inhibit cellular proliferation by inducing a G(2)/M phase cell cycle arrest. Reovirus-induced G(2)/M phase arrest requires the viral S1 gene-encoded sigma1s nonstructural protein. The G(2)-to-M transition represents a cell cycle checkpoint that is regulated by the kinase p34(cdc2). We now report that infection with serotype 3 reovirus strain Abney, but not serotype 1 reovirus strain Lang, is associated with inhibition and hyperphosphorylation of p34(cdc2). The sigma1s protein is necessary and sufficient for inhibitory phosphorylation of p34(cdc2), since a viral mutant lacking sigma1s fails to hyperphosphorylate p34(cdc2) and inducible expression of sigma1s is sufficient for p34(cdc2) hyperphosphorylation. These studies establish a mechanism by which reovirus can perturb cell cycle regulation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • CDC2 Protein Kinase / physiology*
  • Cell Cycle*
  • Cell Line
  • Mammalian orthoreovirus 3 / physiology*
  • Mice
  • Reoviridae Infections / pathology
  • Reoviridae Infections / virology*
  • Signal Transduction
  • Viral Nonstructural Proteins / physiology*
  • Virus Replication

Substances

  • Viral Nonstructural Proteins
  • sigma 1NS protein, Reovirus
  • CDC2 Protein Kinase