Gene therapy approaches to sensitization of human prostate carcinoma to cisplatin by adenoviral expression of p53 and by antisense jun kinase oligonucleotide methods

Methods Mol Biol. 2001:175:495-520. doi: 10.1385/1-59259-235-X:495.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Adenoviruses, Human*
  • Animals
  • Antineoplastic Agents / pharmacology*
  • Cisplatin / pharmacology*
  • Gene Expression
  • Genetic Therapy
  • Genetic Vectors*
  • Humans
  • JNK Mitogen-Activated Protein Kinases
  • Male
  • Mitogen-Activated Protein Kinases / genetics*
  • Oligonucleotides, Antisense
  • Prostatic Neoplasms / drug therapy
  • Prostatic Neoplasms / therapy*
  • Tumor Suppressor Protein p53 / genetics*

Substances

  • Antineoplastic Agents
  • Oligonucleotides, Antisense
  • Tumor Suppressor Protein p53
  • JNK Mitogen-Activated Protein Kinases
  • Mitogen-Activated Protein Kinases
  • Cisplatin