Abstract
Human teratocarcinoma cells (Tera-2) deprived of serum undergo programmed cell death which can be counteracted by simultaneous addition of IGF-I or IGF-II. This protective effect of IGFs was specific in the sense that both addition of IGF-binding protein-2, and blocking of the IGF-type I receptor by a specific antibody, both resulted in an increased apoptotic rate.
Copyright 2001 Academic Press.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antibodies / pharmacology*
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Apoptosis / drug effects*
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Cell Count
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Cell Division / drug effects
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Dose-Response Relationship, Drug
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Humans
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Insulin-Like Growth Factor I / pharmacology
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Insulin-Like Growth Factor II / pharmacology*
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Receptor, IGF Type 1 / immunology*
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Teratocarcinoma / pathology*
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Teratocarcinoma / prevention & control
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Time Factors
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Tumor Cells, Cultured / cytology
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Tumor Cells, Cultured / drug effects
Substances
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Antibodies
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Insulin-Like Growth Factor I
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Insulin-Like Growth Factor II
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Receptor, IGF Type 1