Disruption in cytokine and chemokine production by T-cells in vertically HIV-1-infected children

Acta Paediatr. 2001 Sep;90(9):989-97. doi: 10.1080/080352501316978057.

Abstract

This study measured cytokine production by mitogen-stimulated peripheral blood mononuclear cells (PBMCs) from 55 human immunodeficiency virus (HIV)-infected children born to HIV-infected mothers, and compared it with vertically exposed but uninfected age-matched children. A significant defect was observed in Th1 cytokine production [interferon-gamma and interleukin-2 (IL-2)] in HIV-infected children compared with controls, but without a concomitant increase in Th2 cytokines. Indeed, IL-5 and IL-10 production was even lower in HIV-infected children than in controls, with the decrease in IL-5 being the best predictive marker of immunodeficiency. In addition, an increased release of tumour necrosis factor-alpha (TNF-alpha) that correlated well with CD4+ levels, and a positive correlation of the TNF-alpha/IL-10 ratio with disease progression was observed. A correlation between AIDS-free status and higher %CD4+ and %CD8+ T-lymphocytes and RANTES (regulated on activation, normal T-cell expressed and secreted) production was also found.

Conclusion: A dysfunctional cytokine production of PBMCs was observed in HIV-infected children in both Th1 and Th2 cytokines due to quantitative and qualitative defects induced by HIV-1. An important observation was an increased RANTES production associated with viral isolates of NSI/R5 phenotype and S/L kinetics.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • CD4-CD8 Ratio
  • Chemokines / biosynthesis
  • Child
  • Child, Preschool
  • HIV Infections / immunology*
  • HIV Infections / transmission
  • HIV-1*
  • Humans
  • Infectious Disease Transmission, Vertical*
  • Interferons / biosynthesis*
  • Interferons / blood
  • Interleukins / biosynthesis*
  • Interleukins / blood
  • Monocytes / metabolism
  • Predictive Value of Tests
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism*
  • Tumor Necrosis Factor-alpha / biosynthesis*
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Chemokines
  • Interleukins
  • Tumor Necrosis Factor-alpha
  • Interferons