TGF-beta is required for programmed cell death in interdigital webs of the developing mouse limb

Mech Dev. 2002 May;113(2):111-20. doi: 10.1016/s0925-4773(02)00015-1.

Abstract

During limb formation massive cell death in the mesenchyme of the interdigital spaces accompanies the formation of free digits. Members of the transforming growth factor beta (TGF-) superfamily were discussed to play a key role in cell-cell interactions, important in the regulation of programmed cell death (PCD). TGF-beta itself is believed to be involved in epithelial-mesenchymal interactions. Here, we demonstrate that PCD is significantly reduced in interdigital spaces of the developing limbs of Tgfbeta2-/-Tgfbeta3-/- double knockouts. The regression of interdigital webs seems to be doses-dependent as interdigital mesenchyme is at least partly reduced in Tgfbeta2-/-Tgfbeta3+/- mutants, whereas interdigital zones of Tgfbeta2-/-Tgfbeta3-/- double knockouts reveal only minimal signs of regression. We conclude that TGF- is a critical extrinsic regulator of PCD.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis*
  • Caspase 3
  • Caspases / biosynthesis
  • Chondrocytes / metabolism
  • Enzyme Activation
  • Extremities / embryology*
  • Heterozygote
  • Immunohistochemistry
  • In Situ Nick-End Labeling
  • Mesoderm / metabolism
  • Mice
  • Mice, Knockout
  • Phosphorylation
  • Time Factors
  • Transforming Growth Factor beta / genetics*
  • Transforming Growth Factor beta / physiology*
  • Transforming Growth Factor beta2
  • Transforming Growth Factor beta3

Substances

  • Tgfb3 protein, mouse
  • Transforming Growth Factor beta
  • Transforming Growth Factor beta2
  • Transforming Growth Factor beta3
  • Casp3 protein, mouse
  • Caspase 3
  • Caspases