Expression, proteolysis and activation of caspases 6 and 7 during rat C6 glioma cell apoptosis

Neurosci Lett. 2002 May 10;324(1):33-6. doi: 10.1016/s0304-3940(02)00166-0.

Abstract

Activation of cysteinyl aspartate-specific proteases (caspases) may underlie apoptotic cell death in brain. Terminal, executioner caspases 3, 6 and 7 likely contribute to such cell death in a stimulus- and cell type-specific manner. Here we investigate the processing and activation of caspases 3, 6 and 7 in rat C6 glioma cells induced to undergo apoptosis by staurosporine (STS) treatment as a model of apoptosis in glia. Proteolysis and activation of caspases 3 and 7 as determined by immunoblotting and substrate-specific cleavage assay (DEVDase) preceded caspase-6 proteolysis and increased VEIDase activity following STS treatment. Activation of caspase-6 was paralleled by cleavage of the nuclear envelope protein lamin-A. These results highlight temporal differences in the activation of the triad of executioner caspases 3, 6 and 7 during glial cell apoptosis.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Apoptosis / physiology*
  • Brain / enzymology*
  • Brain / physiopathology
  • Brain Diseases / enzymology*
  • Brain Diseases / physiopathology
  • Caspase 3
  • Caspase 6
  • Caspase 7
  • Caspases / metabolism*
  • Enzyme Inhibitors / pharmacology
  • Fluorescent Antibody Technique
  • Gene Expression Regulation, Enzymologic / physiology*
  • Glioma
  • In Situ Nick-End Labeling
  • Lamin Type A
  • Lamins
  • Neuroglia / enzymology*
  • Neuroglia / pathology
  • Nuclear Proteins / metabolism
  • Peptide Hydrolases / metabolism*
  • Rats
  • Staurosporine / pharmacology
  • Tumor Cells, Cultured

Substances

  • Enzyme Inhibitors
  • Lamin Type A
  • Lamins
  • Nuclear Proteins
  • Peptide Hydrolases
  • Casp3 protein, rat
  • Casp6 protein, rat
  • Caspase 3
  • Caspase 6
  • Caspase 7
  • Caspases
  • Staurosporine