[The high frequency of spontaneous micronuclei observed in lymphocytes of systemic sclerosis patients: preliminary results]

Reumatismo. 2002;54(1):36-9. doi: 10.4081/reumatismo.2002.36.
[Article in Italian]

Abstract

Objective: Aim of the study is to assess the presence of spontaneous chromosome damage in patients affected by limited (lSSc) or diffuse (dSSc) Systemic Slerosis, using the micronucleus (MN) assay.

Methods: We evaluated MN frequency in cultured peripheral lymphocytes of 18 SSc and in a group of 20 healthy controls. Patients were also classified as ACA+, Scl70+, FAN+ according to the presence of the specific anti-nuclear antibodies. We also explored the hypothesis that the extent of cytogenetic alteration might be related to the severity of the pathological condition and/or to the immunological profile.

Results: Compared to controls, the patient group as a whole showed significantly higher MN frequencies (10.8+/-4.5 vs. 27.8+/-13.7, p<0.001). No correlation was found between spontaneous chromosome damage and severity of the disease, being MN frequency 33.1+/-17.0 and 19.8+/-2.7 in lSSc and dSSc, respectively. Interestingly, ACA+ subjects displayed the highest MN frequency (36.9+/-15.0), as compared to patients with different antibody pattern (Scl70+, FAN+; 19.7+/-8.2).

Conclusions: Our results confirm the presence of chromosomal damage in circulating lymphocytes of SSc patients and would suggest a key role of antibodies to the centromere in determining the observed cytogenetic anomalies.

MeSH terms

  • Aged
  • Antibodies, Antinuclear / blood
  • Antibody Specificity
  • Autoantigens / immunology
  • Autoimmune Diseases / blood
  • Autoimmune Diseases / genetics*
  • Centromere / immunology
  • Chromosome Aberrations
  • DNA Topoisomerases, Type I
  • Female
  • Humans
  • Lymphocytes / ultrastructure
  • Male
  • Micronucleus Tests*
  • Middle Aged
  • Nuclear Proteins / immunology
  • Scleroderma, Systemic / blood
  • Scleroderma, Systemic / genetics*

Substances

  • Antibodies, Antinuclear
  • Autoantigens
  • Nuclear Proteins
  • Scl 70 antigen, human
  • DNA Topoisomerases, Type I