Oxysterol-induced apoptosis of vascular smooth muscle cells is reduced by HMG-CoA reductase inhibitor, pravastatin

J Atheroscler Thromb. 2002;9(1):65-71. doi: 10.5551/jat.9.65.

Abstract

We investigated the mechanism by which 7-ketocholesterol damages vascular smooth muscle cells and the protective effect of the hydroxymethyl glutary CoA reductase inhibitor, pravastatin on it. When 7-ketocholesterol (50 micromol/L) was added to cultured human vascular smooth muscle cells, the extent of cell detachment increased and terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling was positive. DNA extracted from the smooth muscle cells exposed to 7-ketocholesterol showed a ladder pattern on agarose electrophoresis. The fragmented DNA also increased in smooth muscle cells incubated with 7-ketocholesterol dose-dependently. In the presence of pravastatin, the cell detachment induced by 7-ketocholesterol was inhibited and the amount of fragmented DNA decreased significantly. These effects of pravastatin were inhibited by mevalonate. The results suggest that 7-ketocholesterol-induced apoptosis of vascular smooth muscle cells is inhibited by pravastatin, and mevalonate acts as a trigger of the apoptosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anticholesteremic Agents / pharmacology*
  • Cell Adhesion / drug effects
  • Cells, Cultured
  • Cholesterol / pharmacology
  • Coronary Vessels / cytology
  • DNA / analysis
  • DNA Fragmentation / drug effects*
  • Dose-Response Relationship, Drug
  • Enzyme Inhibitors / toxicity*
  • Humans
  • In Situ Nick-End Labeling
  • Ketocholesterols / toxicity*
  • Mevalonic Acid / pharmacology
  • Muscle, Smooth, Vascular / cytology*
  • Muscle, Smooth, Vascular / drug effects
  • Pravastatin / pharmacology*

Substances

  • Anticholesteremic Agents
  • Enzyme Inhibitors
  • Ketocholesterols
  • DNA
  • Cholesterol
  • Pravastatin
  • 7-ketocholesterol
  • Mevalonic Acid