CARD15/NOD2 analysis in rheumatoid arthritis susceptibility

Rheumatology (Oxford). 2003 Nov;42(11):1380-2. doi: 10.1093/rheumatology/keg383. Epub 2003 Jun 16.

Abstract

Objective: To determine if the mutations in the CARD15/NOD2 gene predisposing to Crohn's disease (CD) contribute also to the genetic susceptibility to rheumatoid arthritis (RA).

Methods: The frequencies of the three commonest mutations of CARD15/NOD2 predisposing to CD (2104C > T, 2722G>C and 3020insC) were determined in 210 RA patients and 227 controls.

Results: Allelic frequencies of the CARD15/NOD2 mutations in RA patients (2104C>T, 2.8%; 2722G>C, 0.9%; and 3020insC, 2.4%) did not differ significantly from the controls (2104C>T, 5.3%; 2722G>C, 0.7%; and 3020insC, 1.1%).

Conclusion: There was no evidence of association between the commonest CD CARD15/NOD2 mutations and RA susceptibility.

Publication types

  • Clinical Trial
  • Controlled Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Arthritis, Rheumatoid / genetics*
  • Carrier Proteins / genetics*
  • Case-Control Studies
  • Gene Frequency
  • Genetic Predisposition to Disease*
  • Genotype
  • Humans
  • Intracellular Signaling Peptides and Proteins*
  • Mutation / genetics*
  • Nod2 Signaling Adaptor Protein
  • Polymorphism, Genetic

Substances

  • Carrier Proteins
  • Intracellular Signaling Peptides and Proteins
  • NOD2 protein, human
  • Nod2 Signaling Adaptor Protein