Abstract
In rat neocortical slices activation of glutamate metabotropic receptors with glutamate, quisqualate and 1S,3R-ACPD caused a spike-evoked afterdepolarization which replaced the normal afterhyperpolarization. In some neocortical neurons the afterdepolarization reached action potential threshold causing prolonged self-sustained firing. These effects of metabotropic receptor activation were similar to the action of muscarinic agonists in the same preparation.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Action Potentials / drug effects
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Animals
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Cerebral Cortex / drug effects
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Cerebral Cortex / metabolism*
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Cycloleucine / analogs & derivatives
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Cycloleucine / pharmacology
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Glutamates / pharmacology
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Glutamic Acid
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In Vitro Techniques
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Microelectrodes
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Neuromuscular Depolarizing Agents / pharmacology
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Neurons / drug effects
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Neurons / metabolism*
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Quisqualic Acid / pharmacology
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Rats
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Receptors, Glutamate / drug effects
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Receptors, Glutamate / metabolism*
Substances
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Glutamates
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Neuromuscular Depolarizing Agents
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Receptors, Glutamate
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Cycloleucine
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1-amino-1,3-dicarboxycyclopentane
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Glutamic Acid
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Quisqualic Acid