Simultaneous transcription of eleven cytokines in human alloreactive T lymphocyte clones after stimulation by phorbol ester and A23187

Eur Cytokine Netw. 1992 Sep-Oct;3(5):477-83.

Abstract

Human alloreactive T lymphocyte clones derived from cells invading a rejected kidney allograft, were analyzed for their ability to transcribe eleven cytokine genes under phorbol ester (PMA) plus calcium ionophore (CaI A 23187) stimulation. In addition to the positive signal previously obtained for IL-2 transcripts, strong specific patterns were seen with cytoplasmic dot hybridizations for IFN gamma and GM-CSF mRNAs in all the 17 clones screened. For the remaining transcripts (IL-3, IL-4, IL-5, IL-6, TNF alpha, LT, M-CSF and HILDA/LIF), these techniques proved to be inadequate. Northern-blots were therefore performed on three clones exhibiting different phenotypes (CD4+ CD8- non cytotoxic, CD4+ CD8- cytotoxic and CD4- CD8+ cytotoxic). Positive specific signal with the eleven probes could be obtained. Nevertheless, the IL-6 message was found only in the helper clone and the TNF alpha transcript appeared at a later time point compared to the other cytokine messages (its maximum expression was observed around 24 hours post-stimulation). In conclusion, we demonstrate that under PMA+CaI activation, one clone is able to simultaneously transcribe at least eleven lymphokine genes. Except, perhaps for IL-6, the pattern of lymphokine transcription did not permit us to distinguish between different lymphocyte subsets.

MeSH terms

  • Blotting, Northern
  • CD4-Positive T-Lymphocytes / drug effects
  • CD4-Positive T-Lymphocytes / metabolism
  • Calcimycin / pharmacology*
  • Cytokines / biosynthesis*
  • Gene Expression Regulation / drug effects
  • Humans
  • Lymphocyte Activation / drug effects
  • Lymphokines / biosynthesis
  • RNA, Messenger / analysis
  • Stimulation, Chemical
  • T-Lymphocyte Subsets / drug effects*
  • T-Lymphocyte Subsets / metabolism
  • T-Lymphocytes, Cytotoxic / drug effects
  • T-Lymphocytes, Cytotoxic / metabolism
  • Tetradecanoylphorbol Acetate / pharmacology*
  • Transcription, Genetic / drug effects*

Substances

  • Cytokines
  • Lymphokines
  • RNA, Messenger
  • Calcimycin
  • Tetradecanoylphorbol Acetate