Expression of endothelial adhesion molecules in vivo. Increased endothelial ICAM-2 expression in lymphoid malignancies

Am J Pathol. 1992 Apr;140(4):763-7.

Abstract

The authors have compared the reactivity of monoclonal antibodies (MAb) directed against endothelial adhesion molecules (ICAM-1, ICAM-2, VCAM-1, ELAM-1) in hyperplastic, nonmalignant, and malignant lymph nodes. The authors demonstrate that the reactivity with ICAM-1 MAb is stronger in the high endothelial venules (HEV) and other smaller vessels (SV) in lymphomas compared with hyperplastic lymph nodes. Similarly, the reactivity of an ICAM-2 MAb (6D5) was shown to be higher in malignant lymph nodes compared with nonmalignant ones. ICAM-2 MAb stained both the HEV and SV. VCAM-1 MAb reacted strongly with germinal centers and its endothelial reactivity was higher in the lymphoma nodes. ELAM-1 MAb stained only faintly some endothelial cells in malignant tissue. These data suggest that besides the known regulatable endothelial adhesion molecules ICAM-1 and VCAM-1, the expression of ICAM-2 can be modified.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antibodies, Monoclonal
  • Antigens, CD / metabolism
  • Cell Adhesion Molecules / metabolism*
  • Endothelium, Vascular / metabolism*
  • Humans
  • Hyperplasia
  • Immunoenzyme Techniques
  • Intercellular Adhesion Molecule-1
  • Lymph Nodes / metabolism
  • Lymph Nodes / pathology
  • Lymphatic Diseases / metabolism*
  • Neoplasms / metabolism
  • Staining and Labeling

Substances

  • Antibodies, Monoclonal
  • Antigens, CD
  • Cell Adhesion Molecules
  • Intercellular Adhesion Molecule-1