Feto-maternal interaction of IGF-I and its binding proteins in fetal growth

Early Hum Dev. 1992 Jun-Jul;29(1-3):187-91. doi: 10.1016/0378-3782(92)90148-a.

Abstract

Elevated levels of maternal IGF-I and IGF-binding proteins IGFBP-1 are regulated by placental hormones rather than pituitary control during pregnancy. Maternal IGF-I promotes fetal growth by stimulating nutrient transfer to fetus through placenta and IGFBP-1 suppresses fetal growth by inhibiting IGF-I binding to receptors in placenta. Since IGF-I and IGFBP-1 are produced in placenta and decidua, respectively, fetal growth is regulated by these substances in the locally formed paracrine system.

MeSH terms

  • Carrier Proteins / physiology*
  • Embryonic and Fetal Development / physiology*
  • Female
  • Humans
  • Insulin-Like Growth Factor Binding Proteins
  • Insulin-Like Growth Factor I / physiology*
  • Maternal-Fetal Exchange*
  • Pregnancy
  • Somatomedins / physiology*

Substances

  • Carrier Proteins
  • Insulin-Like Growth Factor Binding Proteins
  • Somatomedins
  • Insulin-Like Growth Factor I