Two patients with severe corneal disease in KID syndrome

Am J Ophthalmol. 2004 Jan;137(1):181-3. doi: 10.1016/s0002-9394(03)00739-6.

Abstract

Purpose: To report two independent Japanese patients with keratitis, ichthyosis, and deafness (KID) syndrome and severe corneal disorder.

Design: Observational case reports.

Methods: Clinical observation of a 5-year-old boy (Patient 1) and a 64-year-old man (Patient 2) with KID syndrome, presenting prominent corneal diseases. Molecular genetic assessment of the GJB2 gene encoding connexin-26 was performed.

Results: Patient 1 had bilateral diffuse superficial punctuate keratopathy with severe corneal neovascularization. He had a missense mutation of the GJB2 gene. Patient 2 had bilateral corneal stromal keratitis and right corneal ulceration with rupture of the Descemet membrane. He did not have any pathologic mutation of the GJB2 gene. The area of palisades of Vogt was diminished and tear production reduced in both patients. Topical eye drops, and corticosteroid or antibiotics, respectively, relieved them effectively.

Conclusion: The impaired ocular surface regulating system might be a cause of corneal disease in KID syndrome and it can be treated by eye drops.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anti-Bacterial Agents / therapeutic use
  • Child, Preschool
  • Connexin 26
  • Connexins / genetics
  • Corneal Neovascularization / complications
  • Corneal Neovascularization / drug therapy
  • Corneal Neovascularization / genetics
  • Corneal Ulcer / complications*
  • Corneal Ulcer / drug therapy
  • Corneal Ulcer / genetics
  • Glucocorticoids / therapeutic use
  • Hearing Loss, Sensorineural / complications*
  • Hearing Loss, Sensorineural / drug therapy
  • Hearing Loss, Sensorineural / genetics
  • Humans
  • Ichthyosis / complications*
  • Ichthyosis / drug therapy
  • Ichthyosis / genetics
  • Male
  • Middle Aged
  • Mutation, Missense
  • Ophthalmic Solutions
  • Syndrome

Substances

  • Anti-Bacterial Agents
  • Connexins
  • GJB2 protein, human
  • Glucocorticoids
  • Ophthalmic Solutions
  • Connexin 26