Radiosensitization by pan ErbB inhibitor CI-1033 in vitro and in vivo

Clin Cancer Res. 2004 Jan 15;10(2):691-700. doi: 10.1158/1078-0432.ccr-1041-03.

Abstract

Purpose: Overexpression of the ErbB family of receptor tyrosine kinases has been associated with uncontrolled growth of many tumor types and, therefore, presents a promising molecular target for cancer therapy. CI-1033 is a small molecule tyrosine kinase inhibitor that differs from other 4-anilinoquinazolines by being a pan ErbB (instead of epidermal growth factor receptor-specific) irreversible (instead of reversible) inhibitor. Therefore, we investigated the antitumor effect of CI-1033 alone and in combination with ionizing radiation in vitro and in vivo.

Experimental design: We selected three human colon carcinoma cell-lines (LoVo, Caco-2, which express activated epidermal growth factor receptor and ErbB-2 family members, and SW620, which does not), and analyzed the effects of CI-1033 both in vitro and in vivo. For in vivo studies LoVo and Caco-2 cells were implanted s.c. in the flank of nude mice. After the tumor reached approximately 100 mm(3), treatment was initiated with 20 mg/kg of CI-1033 (orally once daily x 5 for 3 successive weeks), radiation treatment (a total of 30 Gy given in 2 Gy once daily x 5 for 3 successive weeks), or a combination of both CI-1033 and radiation treatment.

Results: We found that exposure of LoVo and Caco-2, but not SW620 cells, to CI-1033 in the range of 1-3 micro M could inhibit constitutive signaling by tyrosine kinases, arrest cell growth, inhibit cells in G(1), stimulate expression of p53, and induce apoptosis. The inhibition of cell growth by CI-1033 seemed to produce only minimal radiosensitization in LoVo and Caco-2 cells. In contrast, the combination of CI-1033 and radiation produced significant (P < 0.0005 and P = 0.0002, respectively) and prolonged suppression of tumor growth in both the tumor types when compared with either treatment alone.

Conclusions: These findings suggest that CI-1033 can increase the effectiveness of radiation therapy. The extent of suppression of tyrosine kinase activity by CI-1033, rather than the amount of activity in untreated cells, seemed to be more closely associated with the efficacy of combination treatment.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antineoplastic Agents / pharmacology
  • Apoptosis
  • Bromodeoxyuridine / pharmacology
  • Cell Line, Tumor
  • Cell Survival
  • Coloring Agents / pharmacology
  • DNA / metabolism
  • Dose-Response Relationship, Radiation
  • ErbB Receptors / metabolism
  • Flow Cytometry
  • G1 Phase
  • Humans
  • Immunohistochemistry
  • In Vitro Techniques
  • Mice
  • Mice, Nude
  • Morpholines / pharmacology*
  • Neoplasm Transplantation
  • Oligonucleotide Array Sequence Analysis
  • Precipitin Tests
  • Protein-Tyrosine Kinases / metabolism
  • Quinazolines / pharmacology
  • RNA / metabolism
  • Radiation, Ionizing
  • Radiation-Sensitizing Agents / pharmacology
  • Receptor, ErbB-2 / antagonists & inhibitors*
  • Receptor, ErbB-2 / metabolism*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Time Factors

Substances

  • Antineoplastic Agents
  • Coloring Agents
  • Morpholines
  • Quinazolines
  • Radiation-Sensitizing Agents
  • RNA
  • DNA
  • Canertinib
  • ErbB Receptors
  • Protein-Tyrosine Kinases
  • Receptor, ErbB-2
  • Bromodeoxyuridine