Transcriptional control of TFF3 (intestinal trefoil factor) via promoter binding sites for the nuclear factor kappaB and C/EBPbeta

Peptides. 2004 May;25(5):849-54. doi: 10.1016/j.peptides.2003.11.019.

Abstract

The acute phase response is strictly connected with modulation of gene expression. Transcriptional control of many genes is mediated by binding of diverse transcription factors to cis-acting DNA motifs in the respective promoter sequence. We now describe such regulatory elements for the TFF3 gene coding for a peptide involved in response to gut irritation. TNF-alpha stimulation, which induces NF-kappaB activation, evoked up to 10-fold reduction of TFF3 expression in the colon tumour cell line HT-29. Several consensus binding sites for members of the NF-kappaB transcription factor subunits are located in the 5'-flanking region of TFF3. Mutating these sites was aimed at discovering which one is responsible for NF-kappaB binding and thus regulation of TFF3 expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • CCAAT-Enhancer-Binding Proteins / genetics
  • Electrophoretic Mobility Shift Assay
  • HT29 Cells
  • Humans
  • Mucins / genetics*
  • Muscle Proteins / genetics*
  • Mutation / genetics
  • NF-kappa B / genetics*
  • Peptides
  • Promoter Regions, Genetic*
  • Transcription Factor RelA
  • Transcription Factors / genetics*
  • Transcriptional Activation / drug effects*
  • Trefoil Factor-3
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • CCAAT-Enhancer-Binding Proteins
  • Mucins
  • Muscle Proteins
  • NF-kappa B
  • Peptides
  • TFF3 protein, human
  • TNF protein, human
  • Transcription Factor RelA
  • Transcription Factors
  • Trefoil Factor-3
  • Tumor Necrosis Factor-alpha