HLA-A and -B allele expression and ability to develop anti-Gag cross-clade responses in subtype C HIV-1-infected Ethiopians

Hum Immunol. 2004 Jun;65(6):648-59. doi: 10.1016/j.humimm.2004.02.031.

Abstract

A cohort of 35 human immunodeficiency virus type 1 (HIV-1) subtype C-infected Ethiopians was studied to define the HLA phenotype in all 35 subjects and highly conserved Gag protein regions involved in cross-clade cell-mediated immunity. Full-length Gag virus sequences were determined in 15 individuals. CD8 cell-mediated immune responses were detected by interferon-gamma ELISpot assay. HLA-A*03, -B*49, and -B*57 allelic frequencies were relatively higher than in other African populations. Anti-p17 (aa 1-60) CD8+ were detectable in the highest number of individuals. Anti-p17 (aa 1-60 and 51-110) cross-clade responses against subtype B and C were detected in 50% of the tested subjects. The p24 KF11 (aa 162-172) epitope was found to be immunodominant among the HLA-B*5703--positive individuals. These data represent the first report of correlating HLA phenotype and HIV-specific cell-mediated immune responses among infected Ethiopians and may be useful in designing cytotoxic T lymphocyte-inducing vaccines for this part of Africa.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • AIDS Vaccines / immunology
  • Ethiopia
  • Female
  • Gene Products, gag / genetics
  • Gene Products, gag / immunology*
  • HIV Infections / genetics*
  • HIV Infections / immunology
  • HIV Infections / prevention & control
  • HIV-1 / genetics
  • HIV-1 / immunology*
  • HLA-A Antigens / genetics*
  • HLA-A Antigens / immunology
  • HLA-B Antigens / genetics*
  • HLA-B Antigens / immunology
  • Humans
  • Immunity, Cellular / genetics*
  • Immunity, Cellular / immunology
  • Interferon-gamma / immunology
  • Male
  • Phenotype
  • T-Lymphocytes, Cytotoxic / immunology

Substances

  • AIDS Vaccines
  • Gene Products, gag
  • HLA-A Antigens
  • HLA-B Antigens
  • Interferon-gamma