JNK regulates expression and autocrine signaling of TGF-beta1

Mol Cell. 2004 Jul 23;15(2):170-1. doi: 10.1016/j.molcel.2004.07.001.

Abstract

TGF-beta1 can induce its own expression by an autoregulatory mechanism. In this issue of Molecular Cell, Ventura et al. (2004) show, using JNK-deficient fibroblasts, that JNK negatively regulates TGF-beta1 gene expression and, consequently, autocrine TGF-beta signaling. The results highlight the contributions of JNK signaling in the control of TGF-beta expression and signaling, and of autocrine TGF-beta signaling in the JNK-regulated changes in cell behavior.

Publication types

  • Comment
  • Editorial

MeSH terms

  • Animals
  • Autocrine Communication*
  • Fibroblasts / metabolism
  • JNK Mitogen-Activated Protein Kinases
  • Mice
  • Mice, Knockout
  • Mitogen-Activated Protein Kinases / physiology*
  • Signal Transduction*
  • Transforming Growth Factor beta / metabolism*
  • Transforming Growth Factor beta1

Substances

  • Tgfb1 protein, mouse
  • Transforming Growth Factor beta
  • Transforming Growth Factor beta1
  • JNK Mitogen-Activated Protein Kinases
  • Mitogen-Activated Protein Kinases