Class I histone deacetylase-selective novel synthetic inhibitors potently inhibit human tumor proliferation

Clin Cancer Res. 2004 Aug 1;10(15):5271-81. doi: 10.1158/1078-0432.CCR-03-0709.

Abstract

We have developed previously a class of synthetic hybrid histone deacetylase (HDAC) inhibitors, which were built from hydroxamic acid of trichostatin A and pyridyl ring of MS-275. In this study we evaluated the antitumor effects of these novel hybrid synthetic HDAC inhibitors, SK-7041 and SK-7068, on human cancer cells. Both SK-7041 and SK-7068 effectively inhibited cellular HDAC activity at nanomolar concentrations and induced the time-dependent hyperacetylation of histones H3 and H4. These HDAC inhibitors preferentially inhibited the enzymatic activities of HDAC1 and HDAC2, as compared with the other HDAC isotypes, indicating that class I HDAC is the major target of SK-7041 and SK-7068. We found that these compounds exhibited potent antiproliferative activity against various human cancer cells in vitro. Growth inhibition effect of SK-7041 and SK-7068 was related with the induction of aberrant mitosis and apoptosis in human gastric cancer cells. Both compounds induced the accumulation of cells at mitosis after 6 h of treatment, which was demonstrated by accumulation of tetraploid cells, lack of G(2) cyclin/cyclin-dependent kinase inactivation, and higher mitotic index. After 12 h of treatment, apoptotic cells were increased through mitochondrial and caspase-mediated pathway. Finally, in vivo experiment showed that SK-7041 or SK-7068 was found to reduce the growth of implanted human tumors in nude mice. Therefore, based on isotype specificity and antitumor activity, SK-7041 and SK-7068 HDAC inhibitors are expected to be promising anticancer therapeutic agents and need additional clinical development.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amides / chemistry
  • Amides / pharmacology*
  • Animals
  • Antineoplastic Agents / pharmacology*
  • Apoptosis
  • Biphenyl Compounds / chemistry
  • Biphenyl Compounds / pharmacology*
  • Cell Line, Tumor
  • Cell Nucleus / metabolism
  • Cell Proliferation / drug effects
  • DNA / chemistry
  • Dose-Response Relationship, Drug
  • Enzyme Inhibitors / pharmacology*
  • Histone Deacetylase 1
  • Histone Deacetylase 2
  • Histone Deacetylase Inhibitors*
  • Histones / metabolism
  • Humans
  • Immunoblotting
  • Immunoprecipitation
  • Inhibitory Concentration 50
  • Mice
  • Mice, Nude
  • Mitosis
  • Models, Chemical
  • Neoplasm Transplantation
  • Neoplasms / drug therapy*
  • Pyrrolidines / chemistry
  • Pyrrolidines / pharmacology*
  • Repressor Proteins / antagonists & inhibitors
  • Stomach Neoplasms / drug therapy
  • Time Factors

Substances

  • Amides
  • Antineoplastic Agents
  • Biphenyl Compounds
  • Enzyme Inhibitors
  • Histone Deacetylase Inhibitors
  • Histones
  • Pyrrolidines
  • Repressor Proteins
  • SK-7041
  • SK-7068
  • DNA
  • HDAC1 protein, human
  • Hdac2 protein, mouse
  • Histone Deacetylase 1
  • Histone Deacetylase 2