IL-15 activates telomerase and minimizes telomere loss and may preserve the replicative life span of memory CD8+ T cells in vitro

J Immunol. 2005 Apr 1;174(7):4019-24. doi: 10.4049/jimmunol.174.7.4019.

Abstract

The preservation of the replicative life span of memory CD8(+) T cells is vital for long-term immune protection. Although IL-15 plays a key role in the homeostasis of memory CD8(+) T cells, it is unknown whether IL-15 regulates the replicative life span of memory CD8(+) T cells. In this study, we report an analysis of telomerase expression and telomere length in human memory phenotype CD8(+) T cells maintained by IL-15 in vitro. We demonstrate that IL-15 is capable of activating telomerase in memory CD8(+) T cells via Jak3 and PI3K signaling pathways. Furthermore, IL-15 induces a sustained level of telomerase activity over long periods of time, and in turn minimizes telomere loss in memory CD8(+) T cells after substantial cell divisions. These findings suggest that IL-15 activates stable telomerase expression and compensates telomere loss in memory phenotype CD8(+) T cells, and that telomerase may play an important role in memory CD8(+) T cell homeostasis.

MeSH terms

  • CD8-Positive T-Lymphocytes / enzymology
  • CD8-Positive T-Lymphocytes / physiology*
  • Cellular Senescence / immunology*
  • Enzyme Activation
  • Humans
  • Immunologic Memory*
  • Interleukin-15 / physiology*
  • Janus Kinase 3
  • Phosphatidylinositol 3-Kinases / metabolism
  • Protein-Tyrosine Kinases / metabolism
  • Signal Transduction
  • Telomerase / metabolism*
  • Telomere / metabolism*
  • Telomere / ultrastructure

Substances

  • Interleukin-15
  • Phosphatidylinositol 3-Kinases
  • Protein-Tyrosine Kinases
  • JAK3 protein, human
  • Janus Kinase 3
  • Telomerase