Role of HdeA in acid resistance and virulence in Brucella abortus 2308

Vet Microbiol. 2005 May 20;107(3-4):307-12. doi: 10.1016/j.vetmic.2005.01.018.

Abstract

Two-dimensional gel electrophoretic analysis of cell lysates suggests that stationary phase production of wild-type levels of an ortholog of the low pH dependent chaperone HdeA in Brucella abortus 2308 during growth in a minimal medium requires the presence of the RNA binding protein Hfq. Although mutational analysis demonstrated that HdeA contributes to acid resistance in this bacterium, this protein is not required for wild-type virulence in the BALB/c mouse model. These experimental findings indicate that the brucellae rely upon additional gene products to resist the acidic conditions they encounter in the phagosomal compartment of host macrophages.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Brucella abortus / genetics
  • Brucella abortus / growth & development
  • Brucella abortus / pathogenicity
  • Brucella abortus / physiology*
  • Brucellosis / microbiology*
  • Electrophoresis, Gel, Two-Dimensional
  • Host Factor 1 Protein / physiology*
  • Hydrogen-Ion Concentration
  • Mice
  • Mice, Inbred BALB C
  • Molecular Sequence Data
  • Sequence Alignment
  • Virulence

Substances

  • Host Factor 1 Protein