Ischemia-induced changes of platelet endothelial cell adhesion molecule-1 in the hippocampal CA1 region in gerbils

Brain Res. 2005 Jun 28;1048(1-2):251-7. doi: 10.1016/j.brainres.2005.04.049.

Abstract

We observed chronological changes of platelet endothelial cell adhesion molecule-1 (PECAM-1), final mediator of neutrophil transendothelial migration, immunoreactivity, and protein level in the gerbil hippocampus proper after 5 min of transient ischemia. One day after ischemic insult, PECAM-1 immunoreactivity and protein level increased slightly in the hippocampus proper. Thereafter, PECAM-1 immunoreactivity and protein level increased significantly in the hippocampus proper by 4 days after ischemic insult. Especially, PECAM-1 in the hippocampal CA1 region was higher than that in the CA2/3 region. Five days after ischemic insult, PECAM-1 immunoreactivity decreased compared to the 4 days post-ischemic group. However, the RNA levels of PECAM-1 in the hippocampus proper were significantly decreased in the 4 days post-ischemic groups compared to that in the sham-operated group. This result suggests that the increase of PECAM-1 and decrease of PECAM-1 RNA in the CA1 region 4 days after ischemia may be associated with transmigration of neurotrophil.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Northern / methods
  • Blotting, Western / methods
  • Cell Count / methods
  • Gerbillinae
  • Hippocampus / metabolism*
  • Hippocampus / pathology
  • Immunohistochemistry / methods
  • Ischemia / metabolism*
  • Ischemia / pathology
  • Male
  • Platelet Endothelial Cell Adhesion Molecule-1 / genetics
  • Platelet Endothelial Cell Adhesion Molecule-1 / metabolism*
  • RNA, Messenger / metabolism
  • Reperfusion / methods
  • Reverse Transcriptase Polymerase Chain Reaction / methods
  • Time Factors

Substances

  • Platelet Endothelial Cell Adhesion Molecule-1
  • RNA, Messenger