HIV-inducing factor in cervicovaginal secretions is associated with bacterial vaginosis in HIV-1-infected women

J Acquir Immune Defic Syndr. 2005 Jul 1;39(3):340-6. doi: 10.1097/01.qai.0000146599.47925.e0.

Abstract

Objective: Certain cervicovaginal lavage (CVL) fluid samples obtained from HIV-1-infected and uninfected women stimulate in vitro HIV-1 replication. This activity, HIV-inducing factor (HIF), changes when CVL fluid is heated. We sought to confirm a previous observation that HIF was associated with bacterial vaginosis (BV).

Methods: HIF was measured in unheated and heated CVL fluid obtained from HIV-1-infected women and compared with the presence of BV by Nugent scores, other genital tract conditions, and cervicovaginal HIV-1 shedding.

Results: Among the 295 women studied, 54% of CVL samples had HIF activity and 21% showed heat-stable HIF activity. In adjusted logistic regression, heat-stable HIF was associated with BV (odds ratio [OR]=51.7, 95% confidence interval [CI]: 5.0, 530.7) and with intermediate flora (OR=43.3, 95% CI: 3.6, 521.1); heat-labile HIF was not associated with BV. Neither heat-stable nor heat-labile HIF was associated with other cervicovaginal conditions nor, after controlling for plasma viral load, with genital tract HIV-1 shedding.

Conclusion: We confirmed the association of HIF with BV and attribute it to the heat-stable component. Heat-stable activity is also associated, although less strongly, with intermediate vaginal flora. We propose that heat-stable HIF is a result of products of BV-associated bacteria.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adolescent
  • Adult
  • Cervix Uteri / metabolism
  • Cervix Uteri / virology
  • Female
  • HIV Infections / complications*
  • HIV Infections / transmission
  • HIV Infections / virology
  • HIV-1* / physiology
  • Hot Temperature
  • Humans
  • In Vitro Techniques
  • Vagina / metabolism
  • Vagina / virology
  • Vaginosis, Bacterial / complications*
  • Vaginosis, Bacterial / physiopathology
  • Virus Replication