CARD15/NOD2 polymorphisms do not explain concordance of Crohn's disease in Swedish monozygotic twins

Dig Liver Dis. 2005 Oct;37(10):768-72. doi: 10.1016/j.dld.2005.05.005.

Abstract

Background: CARD15/NOD2 polymorphisms are associated with Crohn's disease. There is a high concordance for disease and disease phenotype in monozygotic twin pairs with Crohn's disease.

Aim: We studied CARD15/NOD2 polymorphisms in a Swedish, population-based cohort of monozygotic twins with Crohn's disease to assess whether these variants explain disease concordance.

Subjects and methods: Twenty-nine monozygotic twin pairs (concordant n=9, discordant n=20) with Crohn's disease and 192 healthy controls were investigated for the CARD15/NOD2 variants Arg702Trp, Gly908Arg and Leu1007fsinsC.

Results: CARD15/NOD2 mutations were found in 5/38 (13%) twins with Crohn's disease, corresponding to a total allele frequency of 6.6%. Only 2/9 concordant twin pairs carried any of the variants and the remaining seven were wild type genotype. The total allele frequency was 4.4 times higher (95% confidence interval 1.0-21.5, p=0.06) in concordant twins than in discordant ones, 11.1% versus 2.5%. In healthy controls the total allele frequency was 2.6%.

Conclusions: CARD15/NOD2 polymorphisms contribute but do not alone explain concordance of Crohn's disease in monozygotic twins and, at least in a Swedish population, other polymorphisms are required. The low occurrence of CARD15/NOD2 mutations in the study and other Northern European populations suggests that these variants are of less importance in Northern Europe.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Arginine
  • Case-Control Studies
  • Child
  • Crohn Disease / genetics*
  • Female
  • Frameshift Mutation
  • Gene Frequency
  • Genetic Predisposition to Disease
  • Genotype
  • Glycine
  • Humans
  • Intracellular Signaling Peptides and Proteins / genetics*
  • Leucine
  • Male
  • Middle Aged
  • Nod2 Signaling Adaptor Protein
  • Polymorphism, Genetic*
  • Sweden / epidemiology
  • Tryptophan
  • Twins, Monozygotic / genetics*

Substances

  • Intracellular Signaling Peptides and Proteins
  • NOD2 protein, human
  • Nod2 Signaling Adaptor Protein
  • Tryptophan
  • Arginine
  • Leucine
  • Glycine