Expression, synaptic localization, and developmental regulation of Ack1/Pyk1, a cytoplasmic tyrosine kinase highly expressed in the developing and adult brain

J Comp Neurol. 2005 Sep 19;490(2):119-32. doi: 10.1002/cne.20656.

Abstract

Cytosolic tyrosine kinases play a critical role both in neural development and in adult brain function and plasticity. Here we isolated a cDNA with high homology to human Ack1 and mouse Tnk2. This cDNA directs the expression of a 125-kD protein that can be autophosphorylated in tyrosines. Initially, this clone was named Pyk1 for proline-rich tyrosine kinase (Lev et al., 1995); however, since it corresponds to the mouse homolog of Ack1, here we called it Ack1/Pyk1. In this study we show that Ack1/Pyk1 mRNA and protein is highly expressed in the developing and adult brain. The highest levels of Ack1/Pyk1 expression were detected in the hippocampus, neocortex, and cerebellum. Electron microscopy studies showed that Ack1/Pyk1 protein is expressed in these regions both at dendritic spines and presynaptic axon terminals, indicating a role in synaptic function. Furthermore, we demonstrate that Ack1/Pyk1 mRNA levels are strongly upregulated by increased neural activity, produced by intraperitoneal kainate injections. During development, Ack1/Pyk1 was also expressed in the proliferative ventricular zones and in postmitotic maturing neurons. In neuronal cultures, Ack1/Pyk1 was detected in developing dendrites and axons, including dendritic tips and growth cones. Moreover, Ack1/Pyk1 colocalized with Cdc42 GTPase in neuronal cultures and coimmunoprecipitated with Cdc42 in HEK 293T cells. Altogether, our findings indicate that Ack1/Pyk1 tyrosine kinase may be involved both in adult synaptic function and plasticity and in brain development.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Newborn
  • Blotting, Northern / methods
  • Blotting, Western / methods
  • Brain / cytology
  • Brain / embryology
  • Brain / growth & development
  • Brain / metabolism*
  • Cells, Cultured
  • Cloning, Molecular / methods
  • Embryo, Mammalian
  • Excitatory Amino Acid Agonists / pharmacology
  • Gene Expression Regulation, Developmental / drug effects
  • Gene Expression Regulation, Developmental / physiology*
  • Glial Fibrillary Acidic Protein / metabolism
  • Humans
  • Immunohistochemistry / methods
  • Immunoprecipitation / methods
  • In Situ Hybridization / methods
  • Kainic Acid / pharmacology
  • Mice
  • Microscopy, Immunoelectron / methods
  • Microtubule-Associated Proteins / metabolism
  • Neurons / drug effects
  • Neurons / metabolism
  • Neurons / ultrastructure
  • Phosphoamino Acids / metabolism
  • Phosphorylation
  • Presynaptic Terminals / metabolism
  • Presynaptic Terminals / ultrastructure
  • Protein-Tyrosine Kinases / biosynthesis
  • Protein-Tyrosine Kinases / genetics
  • Protein-Tyrosine Kinases / metabolism*
  • RNA, Messenger / biosynthesis
  • Synapses / metabolism*
  • Synapses / ultrastructure
  • Time Factors
  • Tubulin / metabolism
  • cdc42 GTP-Binding Protein / metabolism

Substances

  • Excitatory Amino Acid Agonists
  • Glial Fibrillary Acidic Protein
  • Microtubule-Associated Proteins
  • Mtap2 protein, mouse
  • Phosphoamino Acids
  • RNA, Messenger
  • Tubulin
  • beta3 tubulin, mouse
  • Tnk2 protein, mouse
  • Protein-Tyrosine Kinases
  • TNK2 protein, human
  • cdc42 GTP-Binding Protein
  • Kainic Acid