Src and FAK signalling controls adhesion fate and the epithelial-to-mesenchymal transition

Curr Opin Cell Biol. 2005 Oct;17(5):542-7. doi: 10.1016/j.ceb.2005.08.007.

Abstract

Src kinase controls cellular adhesions, including cadherin-based intercellular adhesions and integrin-mediated cell-matrix adhesions. In epithelial cells, Src activation, or increased signalling from migratory growth factor receptors via Src, induces an adhesion switch that enhances dynamic cell-matrix adhesions and migratory capacity while suppressing intercellular contact. Moreover, Src and the associated tyrosine kinase FAK are at the heart of the recently identified crosstalk between integrin- and cadherin-mediated adhesions of epithelial cells, particularly during the epithelial-to-mesenchymal transition.

MeSH terms

  • Animals
  • Cadherins / physiology
  • Cell Adhesion / physiology*
  • Cell Differentiation*
  • Endocytosis / physiology
  • Epithelial Cells / cytology
  • Epithelial Cells / metabolism*
  • Epithelial Cells / physiology
  • Humans
  • Integrins / physiology
  • Mesoderm / cytology
  • Mesoderm / metabolism*
  • Mesoderm / physiology
  • Models, Biological
  • Neoplasms / metabolism
  • Signal Transduction / physiology*
  • src-Family Kinases / metabolism*
  • src-Family Kinases / physiology

Substances

  • Cadherins
  • Integrins
  • src-Family Kinases