MAdCAM-1 is needed for diabetes development mediated by the T cell clone, BDC-2.5

Immunology. 2005 Dec;116(4):525-31. doi: 10.1111/j.1365-2567.2005.02254.x.

Abstract

The NOD-derived islet-reactive CD4(+) T cell clone, BDC-2.5, is able to transfer diabetes to neonatal non-obese diabetic (NOD) mice but is unable to transfer disease to either adult NOD or NOD scid recipients. Transfer of diabetes to adult recipients by BDC-2.5 is only accomplished by cotransfer of CD8(+) T cells from a diabetic donor. To understand why this CD4(+) T cell clone is able to mediate diabetes in neonatal but not the adult recipients we examined the ability of the clone to traffic in the different recipients. Our studies showed that MAdCAM-1 has a very different expression pattern in the neonatal and adult pancreas. Blockade of this addressin prevents the clone from transferring diabetes to neonatal mice, suggesting that the differential pancreatic expression of MAdCAM-1 in neonatal and adult pancreas provides an explanation of the differences in diabetes development.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / immunology
  • Animals
  • Animals, Newborn
  • CD4-Positive T-Lymphocytes / immunology*
  • CD8-Positive T-Lymphocytes / immunology
  • Cell Adhesion Molecules / immunology*
  • Cell Adhesion Molecules / metabolism
  • Clone Cells / immunology
  • Clone Cells / transplantation
  • Diabetes Mellitus, Experimental / immunology*
  • Diabetes Mellitus, Type 1 / immunology*
  • Lymphocyte Transfusion
  • Mice
  • Mice, Inbred NOD
  • Mice, SCID
  • Mucoproteins
  • Pancreas / metabolism

Substances

  • Cell Adhesion Molecules
  • Madcam1 protein, mouse
  • Mucoproteins