Tissue transglutaminase (TG2) protects cardiomyocytes against ischemia/reperfusion injury by regulating ATP synthesis

Cell Death Differ. 2006 Oct;13(10):1827-9. doi: 10.1038/sj.cdd.4401889. Epub 2006 Mar 10.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Triphosphate / biosynthesis*
  • Animals
  • GTP-Binding Proteins / deficiency
  • GTP-Binding Proteins / genetics
  • GTP-Binding Proteins / metabolism*
  • In Vitro Techniques
  • Male
  • Membrane Potentials
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Mitochondria, Heart / metabolism
  • Myocardial Reperfusion Injury / metabolism
  • Myocardial Reperfusion Injury / prevention & control*
  • Myocytes, Cardiac / cytology
  • Myocytes, Cardiac / metabolism*
  • Protein Glutamine gamma Glutamyltransferase 2
  • Receptors, Adrenergic, alpha-1 / deficiency
  • Receptors, Adrenergic, alpha-1 / genetics
  • Receptors, Adrenergic, alpha-1 / metabolism
  • Signal Transduction
  • Transglutaminases / deficiency
  • Transglutaminases / genetics
  • Transglutaminases / metabolism*

Substances

  • Adra1b protein, mouse
  • Receptors, Adrenergic, alpha-1
  • Adenosine Triphosphate
  • Protein Glutamine gamma Glutamyltransferase 2
  • Transglutaminases
  • GTP-Binding Proteins