Th1- and Th2-type cytokines in chronic ocular allergy

Graefes Arch Clin Exp Ophthalmol. 2006 Oct;244(10):1240-5. doi: 10.1007/s00417-006-0285-7. Epub 2006 Mar 15.

Abstract

Background: Previous reports have suggested that Th2-type cytokines are important in the pathogenesis of ocular allergic diseases. The purpose of this study is to measure levels and mRNA expression of Th1- and Th2-type cytokines in patients with active vernal keratoconjunctivitis (VKC) and atopic keratoconjunctivitis (AKC).

Methods: Tear samples and tear-isolated cells were obtained from 9 healthy participants (CT--controls), 28 VKC, and 6 AKC patients. IL-4, IL-13, and interferon gamma (IFNgamma) tear levels were determined by ELISA, and IL-4 and IFNgamma tear cell mRNA expression by RT-PCR. Effects of these cytokines on IL-6 and IL-8 secretion, and on ICAM-1 expression by conjunctival fibroblasts, were evaluated by ELISA and flow cytometry respectively.

Results: Interleukin-4 tear levels were increased in VKC and AKC compared with CT, but only IFNgamma significantly correlated with corneal involvement. An IL-4/13-dominant profile was found in 50% of VKC and in 17% of AKC patients, while a IFNgamma-dominant profile was found in 18% of VKC and in 17% of AKC patients. IL-4 and IFNgamma transcripts were detected in tear cells from 11 out of 12 VKC patients. IFNgamma upregulated expression of ICAM-1 on conjunctival fibroblasts and the secretion of IL-6 and IL-8.

Conclusions: Although both IL-4 and IFNgamma are detected in tears, only IFNgamma levels correlated with disease severity and upregulated ICAM-1 on conjunctival fibroblasts, suggesting the role of IFNgamma in the inflammatory phase of chronic allergic eye diseases.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Cell Culture Techniques
  • Child
  • Child, Preschool
  • Chronic Disease
  • Conjunctivitis, Allergic / genetics
  • Conjunctivitis, Allergic / metabolism*
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Fibroblasts / cytology
  • Fibroblasts / metabolism
  • Humans
  • Intercellular Adhesion Molecule-1 / metabolism
  • Interferon-gamma / genetics
  • Interferon-gamma / metabolism*
  • Interleukin-13 / genetics
  • Interleukin-13 / metabolism*
  • Interleukin-4 / genetics
  • Interleukin-4 / metabolism*
  • Male
  • Middle Aged
  • RNA, Messenger / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Tears / cytology
  • Tears / metabolism*
  • Th1 Cells / metabolism*
  • Th2 Cells / metabolism*

Substances

  • Interleukin-13
  • RNA, Messenger
  • Intercellular Adhesion Molecule-1
  • Interleukin-4
  • Interferon-gamma