Regulation of eicosanoid release in human umbilical endothelial cells

Thromb Res. 1991 Jun 15;62(6):685-95. doi: 10.1016/0049-3848(91)90372-4.

Abstract

The concentration dependent influence of adenine nucleotides, thrombin and ionophore A 23187 on eicosanoid-production of human endothelial cells was investigated. The prostaglandin I2 (PGI2)-and thromboxane A2 (TXA2)-release were highest at concentrations of 1 mmol/l and 4 mmol/l adenine nucleotides, respectively. TXA2-release decreased at higher concentrations. Ionophore-induced eicosanoid-formation reached its maximum at 15 mumol/l for both PGI2 and TXA2. Thrombin stimulation resulted in a bell shaped concentration dependency, whereby most TXA2 was released at a concentration of 1 U/ml and most PGI2 at 5 U/ml. Intracellular cAMP showed significant (p less than 0.01) increases stimulating cells with ADP- and ATP-concentrations higher than 500 mumol/l, whereas ionophore and thrombin did hardly affect cAMP levels.

Publication types

  • Comparative Study

MeSH terms

  • 6-Ketoprostaglandin F1 alpha / analysis
  • Adenosine Diphosphate / pharmacology
  • Adenosine Triphosphate / pharmacology
  • Calcimycin / pharmacology
  • Cells, Cultured
  • Cyclic AMP / metabolism
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / metabolism*
  • Epoprostenol / metabolism*
  • Humans
  • Infant, Newborn
  • Secretory Rate / drug effects
  • Stimulation, Chemical
  • Thrombin / pharmacology
  • Thromboxane A2 / metabolism*
  • Thromboxane B2 / analysis
  • Umbilical Veins

Substances

  • Calcimycin
  • Thromboxane B2
  • Thromboxane A2
  • 6-Ketoprostaglandin F1 alpha
  • Adenosine Diphosphate
  • Adenosine Triphosphate
  • Epoprostenol
  • Cyclic AMP
  • Thrombin