Inhibition of potassium-induced release of histamine from mast cells by tetraethylammonium and tetramethylammonium

Agents Actions. 1990 Apr;30(1-2):143-5. doi: 10.1007/BF01969022.

Abstract

In accordance with our previous results, a marked release of histamine (HA) from rat peritoneal mast cells was initiated by 150 mM KCl in the absence of extracellular Ca2+. This release could be reduced by 20-60 mM tetraethylammonium (TEA) or tetramethylammonium (TMA), the non-selective K(+)-channel blockers, Ouabain, the general inhibitor of (Na+ + K+) ATP-ase, failed to produce any changes in this release. The action of TEA discriminated between the initiation of HA release evoked by different agents, producing a blockade of the K(+)-induced but not the 48/80-stimulated HA release. In total, these data suggest the presence of TEA/TMA-sensitive K(+)-channels in the mast cell membrane and their involvement in one of the possible pathways for the initiation of HA release.

MeSH terms

  • Animals
  • Ascitic Fluid / cytology
  • Histamine Release / drug effects*
  • In Vitro Techniques
  • Male
  • Mast Cells / drug effects
  • Mast Cells / metabolism*
  • Ouabain / pharmacology
  • Potassium / pharmacology*
  • Potassium Channels / drug effects
  • Quaternary Ammonium Compounds / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Sodium-Potassium-Exchanging ATPase / antagonists & inhibitors
  • Tetraethylammonium Compounds / pharmacology*
  • p-Methoxy-N-methylphenethylamine / pharmacology

Substances

  • Potassium Channels
  • Quaternary Ammonium Compounds
  • Tetraethylammonium Compounds
  • p-Methoxy-N-methylphenethylamine
  • Ouabain
  • Sodium-Potassium-Exchanging ATPase
  • tetramethylammonium
  • Potassium