Combined renal tubular acidosis and diabetes insipidus in hematological disease

Nat Clin Pract Nephrol. 2007 Mar;3(3):171-5. doi: 10.1038/ncpneph0424.

Abstract

Background: A 39-year-old male with multiple myeloma was admitted for treatment with melphalan and autologous stem cell reinfusion. He presented with hypokalemia and hyperchloremic non-anion-gap metabolic acidosis with a high urinary pH. He also had hypomagnesemia, hypophosphatemia, hypouricemia, proteinuria and glucosuria. The patient subsequently developed polyuria with a low urine osmolality, hypernatremia and, finally, acute renal failure.

Investigations: Physical examination, blood and urine analyses, kidney biopsy and tonicity balance.

Diagnosis: Fanconi syndrome with proximal (type II) renal tubular acidosis caused by myeloma kidney. Renal tubular acidosis was complicated by probable nephrogenic diabetes insipidus and acute renal failure.

Management: Potassium supplementation, sodium bicarbonate therapy, intravenous fluid therapy and dialysis.

Publication types

  • Case Reports

MeSH terms

  • Acidosis, Renal Tubular / chemically induced
  • Acidosis, Renal Tubular / complications*
  • Acidosis, Renal Tubular / etiology*
  • Acute Kidney Injury / complications
  • Acute Kidney Injury / pathology
  • Adult
  • Amphotericin B / adverse effects
  • Amphotericin B / therapeutic use
  • Antifungal Agents / adverse effects
  • Antifungal Agents / therapeutic use
  • Aspergillosis / complications
  • Aspergillosis / drug therapy
  • Diabetes Insipidus, Nephrogenic / chemically induced
  • Diabetes Insipidus, Nephrogenic / complications*
  • Fanconi Syndrome / etiology
  • Hematologic Diseases / complications*
  • Humans
  • Male
  • Multiple Myeloma / complications
  • Myelodysplastic Syndromes / complications
  • Myelodysplastic Syndromes / therapy

Substances

  • Antifungal Agents
  • Amphotericin B