Regulation of Na+/Ca2+ exchange current in the normal and failing heart

Ann N Y Acad Sci. 2007 Mar:1099:361-72. doi: 10.1196/annals.1387.065.

Abstract

Cardiac NCX is modulated by diverse regulatory elements. Although there is consensus about the regulatory function of Na+ and Ca2+ and other elements, for example, ATP, there is still a controversial debate about the functional role of cyclic nucleotides and protein kinases. Future studies should focus on that topic since disturbances of cAMP/cGMP concentration and kinase activity may lead to severe functional disorders in the diseased heart. S100A1 is presumably a novel regulator of NCX.

Publication types

  • Review

MeSH terms

  • Calcium / metabolism
  • Cardiac Output, Low / physiopathology*
  • Cells, Cultured
  • Heart / physiology*
  • Humans
  • Protein Kinases / metabolism
  • Receptors, Adrenergic, beta / physiology
  • Sodium / metabolism
  • Sodium-Calcium Exchanger / physiology*

Substances

  • Receptors, Adrenergic, beta
  • Sodium-Calcium Exchanger
  • Sodium
  • Protein Kinases
  • Calcium