Region-specific reduction of Gbeta4 expression and induction of the phosphorylation of PKB/Akt and ERK1/2 by aging in rat brain

Pharmacol Res. 2007 Oct;56(4):295-302. doi: 10.1016/j.phrs.2007.07.005. Epub 2007 Jul 27.

Abstract

This study was designed to investigate the change of expression level of G protein beta(4) subunit (Gbeta(4)) and the activity of PKB/Akt and ERK1/2 during aging process. Both mRNA and protein levels of Gbeta(4) and the induction of phosphorylation of PKB/Akt and ERK1/2 were examined in nine regions of brain (frontal cortex, striatum, hypothalamus, hippocampus, cerebellum, cerebral cortex, thalamic area, brainstem, and amygdala-septum-preoptic area) from weanling (3-week-old), young (12-week-old), adult (24-week-old), and aged (80- or 96-week-old) rats. Aging process (defined as the comparison of 12-week-old young rats with 96-week-old aged rats in this study) significantly reduced Gbeta(4) mRNA (in all testing regions except striatum and thalamic area) and protein expression (except hypothalamus and hippocampus). Aging process also caused a significant increase in the phosphorylation of both PKB/Akt in striatum, hippocampus, cerebellum and thalamic area, and ERK1/2 in all testing regions. Although the precise relationship between increased activities of PKB/Akt and ERK1/2 by aging and decreased expression of Gbeta(4) remains unknown, current data represent the first demonstration of a change in Gbeta(4) expression with age in rat brain and suggest that an age-related alteration in expression level of Gbeta(4) may, at least in part, play an important role in aging processes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / metabolism*
  • Animals
  • Blotting, Western
  • Brain / anatomy & histology
  • Brain / metabolism*
  • Enzyme Activation
  • GTP-Binding Protein beta Subunits / biosynthesis*
  • GTP-Binding Protein beta Subunits / genetics
  • Male
  • Mitogen-Activated Protein Kinase 1 / genetics
  • Mitogen-Activated Protein Kinase 1 / metabolism*
  • Mitogen-Activated Protein Kinase 3 / genetics
  • Mitogen-Activated Protein Kinase 3 / metabolism*
  • Organ Specificity
  • Phosphorylation
  • Proto-Oncogene Proteins c-akt / genetics
  • Proto-Oncogene Proteins c-akt / metabolism*
  • RNA, Messenger / biosynthesis
  • Rats
  • Rats, Sprague-Dawley
  • Reverse Transcriptase Polymerase Chain Reaction
  • Time Factors

Substances

  • GTP-Binding Protein beta Subunits
  • RNA, Messenger
  • Proto-Oncogene Proteins c-akt
  • Mitogen-Activated Protein Kinase 1
  • Mitogen-Activated Protein Kinase 3