[Role of Rho GTPase in inhibiting metastatic ability of human prostate cancer cell line PC-3 by omega-3 polyunsaturated fatty acid]

Ai Zheng. 2007 Dec;26(12):1281-6.
[Article in Chinese]

Abstract

Background & objective: Recently, researches refer to the influence of polyunsaturated fatty acid (PUFA) on cancer initiation and progression had been highly concerned. This study was to investigate the effects of 2 kinds of omega-3 PUFA, eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), on the metastatic ability of human prostate cancer cell line PC-3, and explore the role of Rho GTPase in inhibiting cancer metastasis by omega-3 PUFA.

Methods: MTT assay was used to determine the effects of omega-3 PUFA on the proliferation of PC-3 cells. Adhesion assay, invasion assay, and migration assay were used to observe the effects of omega-3 PUFA on the metastatic ability of PC-3 cells. Western blot was used to observe the effects of omega-3 PUFA on the expression of RhoA, Rac1, Rac2, and Cdc42 proteins in PC-3 cells. Laser confocal microscopy was used to investigate the effect of omega-3 PUFA on the reorganization of the microfilaments and microtubules marked by immunofluorescent cytochemistry technology.

Results: Both EPA and DHA inhibited the proliferation of PC-3 cells, and the proliferation inhibition rate increased along with the increase of the concentration and treatment time. When treated with 60 mumol/L EPA or DHA for 48 h, the abilities of adhesion, invasion and migration of PC-3 cells were inhibited (P<0.05). omega-3 PUFA significantly suppressed the expression of Rac1, Rac2 and Cdc42 proteins (P<0.05), influenced the distribution and structure of sytoskeletons.

Conclusion: omega-3 PUFA could inhibit the metastatic ability of PC-3 cells through down-regulating the expression of Rho GTPase and inhibiting the cytoskeleton reorganization.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Adhesion / drug effects*
  • Cell Line, Tumor
  • Cell Movement / drug effects
  • Cell Proliferation / drug effects
  • Cytoskeleton / drug effects
  • Docosahexaenoic Acids / pharmacology
  • Dose-Response Relationship, Drug
  • Down-Regulation
  • Eicosapentaenoic Acid / pharmacology
  • Fatty Acids, Omega-3 / pharmacology*
  • Humans
  • Male
  • Neoplasm Invasiveness
  • Prostatic Neoplasms / enzymology
  • Prostatic Neoplasms / pathology*
  • rho GTP-Binding Proteins / metabolism*

Substances

  • Fatty Acids, Omega-3
  • Docosahexaenoic Acids
  • Eicosapentaenoic Acid
  • rho GTP-Binding Proteins