Abstract
In this concise overview, we discuss recent findings concerning a distinct subgroup of acute myeloid/T-lymphoid leukemia. We describe how we identified these leukemias in multiple cohorts of acute myeloid leukemia (AML) using a combination of gene expression profiling and additional analytic approaches, and how we obtained insight in possible mechanisms leading to their phenotype.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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CCAAT-Enhancer-Binding Proteins / genetics*
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CCAAT-Enhancer-Binding Proteins / metabolism
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Gene Expression Profiling
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Gene Expression Regulation, Leukemic*
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Gene Silencing
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Humans
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Leukemia, Myeloid, Acute / genetics*
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Leukemia, Myeloid, Acute / metabolism*
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Leukemia, T-Cell / genetics*
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Leukemia, T-Cell / metabolism*
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Mutation
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Receptor, Notch1 / genetics
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Receptor, Notch1 / metabolism
Substances
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CCAAT-Enhancer-Binding Proteins
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CEBPA protein, human
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NOTCH1 protein, human
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Receptor, Notch1