Anti-inflammatory activity of probiotic Bifidobacterium: enhancement of IL-10 production in peripheral blood mononuclear cells from ulcerative colitis patients and inhibition of IL-8 secretion in HT-29 cells

World J Gastroenterol. 2008 Apr 28;14(16):2511-6. doi: 10.3748/wjg.14.2511.

Abstract

Aim: To determine the anti-inflammatory activity of probiotic Bifidobacteria in Bifidobacteria-fermented milk (BFM) which is effective against active ulcerative colitis (UC) and exacerbations of UC, and to explore the immunoregulatory mechanisms.

Methods: Peripheral blood mononuclear cells (PBMNC) from UC patients or HT-29 cells were co-cultured with heat-killed probiotic bacteria or culture supernatant of Bifidobacterium breve strain Yakult (BbrY) or Bifidobacterium bifidum strain Yakult (BbiY) to estimate the amount of IL-10 or IL-8 secreted.

Results: Both strains of probiotic Bifidobacteria contained in the BFM induced IL-10 production in PBMNC from UC patients, though BbrY was more effective than BbiY. Conditioned medium (CM) and DNA of both strains inhibited IL-8 secretion in HT-29 cells stimulated with TNF-alpha, whereas no such effect was observed with heat-killed bacteria. The inhibitory effect of CM derived from BbiY was greater than that of CM derived from BbrY. DNAs of the two strains had a comparable inhibitory activity against the secretion of IL-8. CM of BbiY induced a repression of IL-8 gene expression with a higher expression of IkappaB-zeta mRNA 4 h after culture of HT-29 cells compared to that in the absence of CM.

Conclusion: Probiotic Bifidobacterium strains in BFM enhance IL-10 production in PBMNC and inhibit IL-8 secretion in intestinal epithelial cells, suggesting that BFM has anti-inflammatory effects against ulcerative colitis.

MeSH terms

  • Bifidobacterium / physiology*
  • Cell Culture Techniques
  • Colitis, Ulcerative / immunology*
  • Culture Media, Conditioned
  • Cytokines / metabolism
  • DNA, Bacterial / metabolism
  • HT29 Cells / physiology*
  • Humans
  • Interleukin-10 / biosynthesis*
  • Interleukin-8 / antagonists & inhibitors*
  • Interleukin-8 / metabolism
  • Leukocytes, Mononuclear / microbiology
  • Leukocytes, Mononuclear / physiology*
  • Probiotics*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • Culture Media, Conditioned
  • Cytokines
  • DNA, Bacterial
  • Interleukin-8
  • Tumor Necrosis Factor-alpha
  • Interleukin-10