Abstract
A new shunt in the phenylalanine biosynthetic pathway to the nonproteinogenic amino acid L-3-cyclohex-2'-enylalanine was exploited in the marine bacterium Salinispora tropica by mutagenesis to allow for the genetic engineering of unnatural derivatives of the potent proteasome inhibitor salinosporamide A (2) such as antiprotealide (1).
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
MeSH terms
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Lactams / chemistry*
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Lactams / metabolism*
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Lactones / chemistry*
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Lactones / metabolism*
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Micromonosporaceae / genetics*
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Molecular Structure
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Protease Inhibitors / chemistry
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Protease Inhibitors / metabolism*
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Proteasome Endopeptidase Complex / chemistry*
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Protein Engineering*
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Pyrroles / chemistry
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Pyrroles / metabolism*
Substances
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Lactams
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Lactones
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Protease Inhibitors
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Pyrroles
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antiprotealide
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marizomib
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Proteasome Endopeptidase Complex