Prognostic significance of TRAIL-R1 and TRAIL-R3 expression in metastatic colorectal carcinomas

Eur J Cancer. 2008 Oct;44(15):2312-8. doi: 10.1016/j.ejca.2008.06.042. Epub 2008 Aug 26.

Abstract

Drug resistance is believed to cause treatment failure in patients with metastatic colorectal carcinoma (CRC). Resistance to chemotherapy can involve different processes, including apoptosis, whose extrinsic pathway is regulated by expression of death-inducing TRAIL-R1 and -R2 and inhibitory TRAIL-R3 and -R4 cell surface receptors. Therefore, we investigated whether variations in their expression could influence the response to 5-Fluorouracil (5-FU) in metastatic CRC. We analysed TRAIL-R 1, -2, -3 and -4 expression by immuno-histochemistry in CRC, using tissue micro arrays, and found that concomitant low/medium TRAIL-R1 and high TRAIL-R3 expression in primary CRC is significantly associated with a poor response to 5-FU-based first-line chemotherapy and with shorter progression-free survival. Specifically, the median progression-free survival was 3.1 months (poor prognostic group) versus 10.1 in the good prognostic group. Thus, the combination of TRAIL-R1 and TRAIL-R3 expression might represent a predictive and prognostic factor of the response to 5-FU-based first-line chemotherapy in patients with metastatic CRC.

Publication types

  • Controlled Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenocarcinoma / drug therapy
  • Adenocarcinoma / metabolism
  • Adenocarcinoma / secondary*
  • Adult
  • Aged
  • Aged, 80 and over
  • Antimetabolites, Antineoplastic / therapeutic use
  • Biomarkers, Tumor / metabolism*
  • Colorectal Neoplasms / drug therapy
  • Colorectal Neoplasms / metabolism*
  • Disease Progression
  • Disease-Free Survival
  • Female
  • Fluorouracil / therapeutic use
  • GPI-Linked Proteins
  • Humans
  • Liver Neoplasms / drug therapy
  • Liver Neoplasms / metabolism
  • Liver Neoplasms / secondary
  • Male
  • Middle Aged
  • Neoplasm Proteins / metabolism
  • Prognosis
  • Receptors, TNF-Related Apoptosis-Inducing Ligand / metabolism*
  • Receptors, Tumor Necrosis Factor, Member 10c
  • Treatment Outcome
  • Tumor Necrosis Factor Decoy Receptors / metabolism

Substances

  • Antimetabolites, Antineoplastic
  • Biomarkers, Tumor
  • GPI-Linked Proteins
  • Neoplasm Proteins
  • Receptors, TNF-Related Apoptosis-Inducing Ligand
  • Receptors, Tumor Necrosis Factor, Member 10c
  • TNFRSF10C protein, human
  • Tumor Necrosis Factor Decoy Receptors
  • Fluorouracil