Abstract
The expression of c-myc mRNA was examined after 4 h of sodium butyrate treatment in a mouse plasmacytoma (MPC) cell line (S194). Steady-state levels of rearranged c-myc mRNA were suppressed by the agent in S194 cells. Run-on assay demonstrated that the suppression of the rearranged c-myc mRNA in the MPC was correlated with the transcriptional downregulation of the gene. The suppression was also accompanied by the reduced DNase I sensitivity of the gene. These findings suggest that the rapid downregulation of c-myc mRNA by sodium butyrate is subject to regulation at the transcriptional level following the alteration of the DNase I sensitive chromatin structure in mouse plasmacytoma cells.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Blotting, Northern
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Butyrates / pharmacology*
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Butyric Acid
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Chromatin / drug effects
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Chromatin / ultrastructure
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DNA, Neoplasm / drug effects
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Deoxyribonuclease I / pharmacology
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Down-Regulation / drug effects
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Gene Expression Regulation, Neoplastic / drug effects
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Gene Rearrangement / physiology
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Genes, myc / drug effects
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Genes, myc / genetics
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Mice
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Mice, Inbred BALB C
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Plasmacytoma / metabolism
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Plasmacytoma / pathology*
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Proto-Oncogene Proteins c-myc / genetics
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Proto-Oncogene Proteins c-myc / metabolism*
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RNA, Messenger / genetics
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RNA, Messenger / metabolism
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Transcription, Genetic / drug effects
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Tumor Cells, Cultured / drug effects
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Tumor Cells, Cultured / metabolism
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Tumor Cells, Cultured / pathology
Substances
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Butyrates
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Chromatin
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DNA, Neoplasm
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Proto-Oncogene Proteins c-myc
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RNA, Messenger
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Butyric Acid
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Deoxyribonuclease I